© Hallberg Independent Research 2012. Updated 2012-05-21
Already in 2005 research done at Huddinge Hospital showed that human cells exposed to mobile phone carrier frequencies got reduced capacity to assimilate insuline through the cell membrane compared to non-exposed cells. I was informed about this research recently but was unable to find published reports on the subject. I was, however, informed on May 2, 2012 about an interesting experiment that showed the strong influence mobile phone radiation has on an important PNS and CNS enzyme. See Abstract.
According to Swedish Health and Welfare Authority (SOS) there are two types of diabetes. One is due to lack of insuline production and it is called insuline-dependent diabetes. The other form does not depend on a too low insuline production, but still there are all characteristics of diabetes. It is called non-insuline-dependent diabetes.
Obviously, if the insuline production is OK but the cells do not get enough of it, the cell membranes may be the logical cause. They won't let enough of insuline penetrate the membranes and we will have a non-insuline-dependent form of diabetes.
I got curious, and wanted to take a closer look at the statistics and trends of diabetes in Sweden. I downloaded data on the annual number of diabetes patients per 100 000 inhabitants in Sweden.
The trend of insuline-dependent diabetes has been improving year by year since 1998 as can be seen in Figure 1.
Figure 1.The rates of insuline-dependent diabetes has been decreasing continuously since 1998.
But the trend of non-insuline-dependent diabetes shows a clear trend break after 2005 and is fast increasing. See Figure 2.
Figure 2. The trend of non-insuline-dependent diabetes was suddenly broken in 2005 and the rate is now fast increasing.
Looking into age-specific trends, we see that the non-insulin depending form of diabetes (E11) is fast increasing both among old and young people. Just click on the graph to enlarge.
|
Figure 3. Age 80+ |
Figure 4. Age 15-29 |
The mortality in E11 increased both for the age-standardised population 0-85+ and the oldest group, 80+. See Figures 5 and 6.
|
Figure 5. Mortality E11: 0-85+ |
Figure 6. Mortality E11: 85+ |
I will ask responsible authorities to see what explanation they will provide. If I get any response, I will let you know.
A professor at the Karolinska Institute explained to me that the increase of prevalence among older people mainly is due to improved survivability (since 2005 according to the graphs). So, the increasing prevalence of diabetes type 2 (E11) among young people might also be a result of suddenly improved treatment and survivability among young patients with diabetes. From year 2005. It would be interesting to know what type of treatment for diabetes that was introduced that year.
